Review article Cleidocranial dysplasia: clinical and molecular genetics

نویسنده

  • Stefan Mundlos
چکیده

Cleidocranial dysplasia (CCD) (MIM 119600) is an autosomal dominant skeletal dysplasia characterised by abnormal clavicles, patent sutures and fontanelles, supernumerary teeth, short stature, and a variety of other skeletal changes. The disease gene has been mapped to chromosome 6p21 within a region containing CBFA1, a member of the runt family of transcription factors. Mutations in the CBFA1 gene that presumably lead to synthesis of an inactive gene product were identified in patients with CCD. The function of CBFA1 during skeletal development was further elucidated by the generation of mutated mice in which the Cbfa1 gene locus was targeted. Loss of one Cbfa1 allele (+/-) leads to a phenotype very similar to human CCD, featuring hypoplasia of the clavicles and patent fontanelles. Loss of both alleles (-/-) leads to a complete absence of bone owing to a lack of osteoblast diVerentiation. These studies show that haploinsuYciency of CBFA1 causes the CCD phenotype. CBFA1 controls diVerentiation of precursor cells into osteoblasts and is thus essential for membranous as well as endochondral bone formation. (J Med Genet 1999;36:177–182)

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تاریخ انتشار 1999